作者: Robin C. Looft-Wilson , Blair S. Ashley , Janelle E. Billig , Madeline R. Wolfert , Lindsay A. Ambrecht
DOI: 10.1152/AJPREGU.00833.2007
关键词: Nitric oxide synthase 、 Internal medicine 、 Nitric oxide 、 Vasodilation 、 Biology 、 Endocrinology 、 Nitric Oxide Synthase Type III 、 Enos 、 Endothelium 、 Hyperhomocysteinemia 、 Mesenteric arteries
摘要: Hyperhomocysteinemia (HHcy) impairs endothelium-dependent vasodilation by increasing reactive oxygen species, thereby reducing nitric oxide (NO·) bioavailability. It is unclear whether reduced expr...