Fat and fatty acid intake and metabolic effects in the human body.

摘要: nants, to form foam cells. There is convincing evidence that cells can progress fibrous atherosclerotic plaques. a large body of scientific evidence, both in animal models and corroborated by human observational intervention studies, showing dietary fat intake causally involved atherogenesis may also influence arterial thrombosis. With regard the experimental feeding studies animals, elevations plasma lipids caused modification result atherosclerosis. These indicate fats high saturated (SFA), monounsaturated (MUFA) trans fatty acids (TFA) promote atherosclerosis [Brown et al., 2007], whereas diets containing oils polyunsaturated (PUFA) inhibit low-fat do not This review focuses on from man influences metabolic factors associated with risk CVD.