Cross‐resistance and biochemical mechanisms of abamectin resistance in the B‐type Bemisia tabaci
作者： Lihua Wang , Yidong Wu
DOI: 10.1111/J.1439-0418.2006.01140.X
关键词: Esterase inhibitor 、 Fipronil 、 Microbiology 、 Biology 、 Emamectin 、 Strain (chemistry) 、 Abamectin 、 Biochemistry 、 Imidacloprid 、 Piperonyl butoxide 、 Cross-resistance
摘要: : To understand the risk of resistance and possible mechanisms to abamectin in B-type Bemisia tabaci (Gennadius) better, a resistant strain B. was selected laboratory cross-resistance pattern were investigated. The NJ-Abm derived from field population (NJ) collected Nanjing, China 2002 with 18 generations selection laboratory. Compared unselected NJ strain, developed 14.5-fold showed significant emamectin benzoate (4.4-fold) imidacloprid (3.4-fold), but no fipronil. oxidase inhibitor piperonyl butoxide (PBO) glutathione S-transferase diethyl maleate (DEM) produced synergism on (with synergistic ratios 3.9- 4.1-fold respectively); however, esterase triphenyl phosphate (TPP) did not act synergistically abamectin. Biochemical analysis confirmed that P450 monooxygenase activity elevated 2.1- 2.0-fold, respectively, compared strain. This indicated enhanced metabolism mediated by is likely be involved
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