作者: Irene S. Horwitz , Albert I. Kaufman
DOI: 10.1016/0006-8993(79)90703-0
关键词: Baroreceptor 、 Reflex 、 Carotid sinus 、 Blood pressure 、 Internal medicine 、 Chemistry 、 Vagus nerve 、 Endocrinology 、 Sweat gland 、 SWEAT 、 Nictitating membrane
摘要: Abstract (1) In chloralose-anesthetized cats, elevation of carotid sinus pressure caused blood pressure, sweat gland potentials and nictitating membrane tension to decrease. (2) The onset recovery the responses usually preceded respective phases (depressor) response; latencies agreed with predicted for neural reflex pathways. (3) were evoked less consistently than depressor response. (4) experiments where only studied: (a) threshold inhibition activity was similar (b) cutting nerves, or blocking efferent glands, eliminated effects on potentials; (c) held constant, decreases in produced by stimulating peripheral end vagus nerve, did not affect potentials. (5) These results indicate that baroreceptors can reflexly modulate sympathetic neurons whose target organs are fundamentally involved regulation.