作者: Robert L. Rosenfield
DOI: 10.1007/978-1-4613-8483-0_6
关键词: Steroid Metabolism 、 Androgen secretion 、 Follicular atresia 、 Congenital adrenal hyperplasia 、 Polycystic ovary 、 Ovarian Hyperandrogenism 、 Internal medicine 、 Luteinizing hormone level 、 Endocrinology 、 Luteinizing hormone 、 Medicine
摘要: We have proposed that the essence of polycystic ovary syndrome (PCOS) is functional ovarian hyperandrogenism (FOH) (Fig. 6.1) (1, 2). Our model proposes FOH initiated by any disorder causes a large increase in intraovarian androgens. This leads to hyperandrogenemia, which responsible for pilosebaceous manifestations syndrome. also promotes process follicular atresia, anovulatory symptoms.