Hyper-Excitability Followed by Functional Quiescence in Neuronal Cells Caused by Insufficient Cellular Energy (ICE): Treatable by Enhancing the Alternative Cellular Energy (ACE) Pathway

摘要: Living organisms derive energy for cellular activities through three primary mechanisms. The first is photosynthesis, which restricted to plants and certain bacteria. It uses in sunlight combine carbon dioxide with water form carbohydrates plus oxygen. second chemical energy, ob-tainable by all from the metabolism of other organic molecules. third mechanism obtaining alternative (ACE) pathway. ACE pathway expressed as an added dynamic (kinetic) quality body’s fluids. results absorption environmental force termed KELEA (kinetic limiting electrostatic attraction). fundamental role presumably pre-vent fusion annihilation electrostatically attracted opposing electrical charges. can loosen hydrogen bonding between fluid benefits living part enabling more efficient biochemical reactions. Cells require a minimal amount remain viable. Additional required undertake specialized functions. Illnesses result if cells have insufficient (ICE) their Since separated charges, it charges comprising membrane potential cells. proposed that depolarization neuronal leads partial release use depolarized cell contribution overall activation Many brain illnesses currently attributed neurodegeneration are explainable cells’ adaptations ICE. likely comprise initial hyper-excitability obtain additional KELEA, followed functional quiescence prior actual death. Clinical recovery during both hyper-excitable hypoactive phases potentially achievable enhancing Furthermore, among restored functions quiescent may be capacity again attract leading sustainable recovery. opportunity exists extended clinical trials involving neurological psychiatric illnesses.