作者: My-Nhan Nguyen , Mark Ziemann , Helen Kiriazis , Yidan Su , Zara Thomas
DOI: 10.1152/AJPHEART.00609.2018
关键词: Fibrosis 、 Galectin-3 、 Internal medicine 、 Cardiomyopathy 、 Cardiac fibrosis 、 Genetically modified mouse 、 Medicine 、 Endocrinology 、 Ventricular remodeling 、 Heart failure 、 Dilated cardiomyopathy
摘要: … Lgals8, and Lgals9, whereas Lgals4 was suppressed. On the nTG background, these galectin genes were not affected by Gal-3 deletion. In TG/KO hearts, both Lgals3 and Lgals9 were …