Is COVID-19 associated thrombosis caused by overactivation of the complement cascade? A literature review.
作者： Alexander Fletcher-Sandersjöö , Bo-Michael Bellander
DOI: 10.1016/J.THROMRES.2020.06.027
关键词: Immunology 、 Thrombosis 、 Platelet 、 Fibrinolysis 、 Coronavirus disease 2019 (COVID-19) 、 Clinical significance 、 Complement system 、 Inflammation 、 Medicine 、 Coronavirus
摘要: Severe acute respiratory syndrome coronavirus 2 is responsible for the current COVID-19 pandemic resulting in an escalating number of cases and fatalities worldwide. Preliminary …
sci-hub.st 参考文章(77)
Umme Amara, Daniel Rittirsch, Michael Flierl, Uwe Bruckner, Andreas Klos, Florian Gebhard, John D. Lambris, Markus Huber-Lang, Interaction between the coagulation and complement system. Advances in Experimental Medicine and Biology. ,vol. 632, pp. 71- 79 ,(2008) , 10.1007/978-0-387-78952-1_6
P J Sims, E M Faioni, T Wiedmer, S J Shattil, Complement proteins C5b-9 cause release of membrane vesicles from the platelet surface that are enriched in the membrane receptor for coagulation factor Va and express prothrombinase activity. Journal of Biological Chemistry. ,vol. 263, pp. 18205- 18212 ,(1988) , 10.1016/S0021-9258(19)81346-7
Nicolas S. Merle, Remi Noe, Lise Halbwachs-Mecarelli, Veronique Fremeaux-Bacchi, Lubka T. Roumenina, Complement System Part II: Role in Immunity. Frontiers in Immunology. ,vol. 6, pp. 257- 257 ,(2015) , 10.3389/FIMMU.2015.00257
T Wiedmer, C T Esmon, P J Sims, On the mechanism by which complement proteins C5b-9 increase platelet prothrombinase activity. Journal of Biological Chemistry. ,vol. 261, pp. 14587- 14592 ,(1986) , 10.1016/S0021-9258(18)66911-X
K K Hamilton, R Hattori, C T Esmon, P J Sims, Complement proteins C5b-9 induce vesiculation of the endothelial plasma membrane and expose catalytic surface for assembly of the prothrombinase enzyme complex. Journal of Biological Chemistry. ,vol. 265, pp. 3809- 3814 ,(1990) , 10.1016/S0021-9258(19)39666-8
R Hattori, K K Hamilton, R P McEver, P J Sims, Complement proteins C5b-9 induce secretion of high molecular weight multimers of endothelial von Willebrand factor and translocation of granule membrane protein GMP-140 to the cell surface. Journal of Biological Chemistry. ,vol. 264, pp. 9053- 9060 ,(1989) , 10.1016/S0021-9258(18)81901-9
Konstantinos Ritis, Michael Doumas, Dimitrios Mastellos, Anastasia Micheli, Stavros Giaglis, Paola Magotti, Stavros Rafail, Georgios Kartalis, Paschalis Sideras, John D. Lambris, A novel C5a receptor-tissue factor cross-talk in neutrophils links innate immunity to coagulation pathways. Journal of Immunology. ,vol. 177, pp. 4794- 4802 ,(2006) , 10.4049/JIMMUNOL.177.7.4794
Johann Wojta, Kurt Huber, Peter Valent, New aspects in thrombotic research: complement induced switch in mast cells from a profibrinolytic to a prothrombotic phenotype. Pathophysiology of Haemostasis and Thrombosis. ,vol. 33, pp. 438- 441 ,(2003) , 10.1159/000083842
M J Polley, R Nachman, The human complement system in thrombin-mediated platelet function. Journal of Experimental Medicine. ,vol. 147, pp. 1713- 1726 ,(1978) , 10.1084/JEM.147.6.1713
M. Cugno, I. Bos, Y. Lubbers, C. Erik Hack, A. Agostoni, In vitro interaction of C1-inhibitor with thrombin. Blood Coagulation & Fibrinolysis. ,vol. 12, pp. 253- 260 ,(2001) , 10.1097/00001721-200106000-00005