Neutrophil-endothelial cell interactions during the development of tolerance to ischaemia/reperfusion in isolated cells.
作者： G. Cepinskas , T. Rui , P. R. Kvietys
DOI: 10.1046/J.1365-201X.2001.00881.X
关键词: Immunology 、 Inflammation 、 In vitro 、 Endothelial stem cell 、 Heat shock protein 、 Cell biology 、 Transcription factor 、 Biology 、 Effector 、 Cell–cell interaction 、 Nitric oxide synthase
摘要: Ischaemia,/reperfusion (I/R) tolerance refers to the phenomenon by which inflammation and associated sequelae induced I/R is ameliorated an challenge imposed 24 h earlier. The development of dependent on synthesis new proteins. In vivo in vitro studies provide support for nitric oxide synthase (NOS), antioxidant enzymes, heat shock proteins (HSPs) as effector Activation nuclear transcription factor, NFκB, appears be a prerequisite tolerance. approaches using anoxia/ reoxygenation (A/R) mimic have provided insights into complexity tolerance, i.e. different cells may use signalling pathways develop A/R influence responses adjacent during process. from genetically altered mice expediting attempts unravel specific mechanisms involved
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