作者: A. Kahn , J. Groswasser , P. Franco , I. Kelmanson , M. Sottiaux
DOI: 10.1016/S1567-424X(09)70178-3
关键词: Cardiology 、 Medicine 、 Anesthesia 、 CIRCULATORY FAILURE 、 Internal medicine 、 Infant mortality 、 Heart rate 、 Sudden infant death 、 Gliosis 、 Sleep in non-human animals 、 Respiratory system
摘要: Publisher Summary Prevention campaigns to avoid risk factors for the occurrence of sudden infant deaths (SID) during sleep have led a significant decrease in number infants dying suddenly and unexpectedly sleep. This chapter suggests model these underlying mechanisms responsible SID address major challenges facing researchers this field. During has significantly more frequent obstructive apneas, less body movements, sympathetic control heart rate, desynchronized relation between vagal cardiac controls by end night, decreased propensity arouse from The respiratory characteristics that would be found future SIDS victims could related an increased susceptibility dysregulations These findings represent delay maturation vital at SID. arousability reveal lesser autoresuscitate case acute or circulatory failure. neurophysiological result postmortem within victims' brain-stem, such as abnormally occurrences brain-stem gliosis, cellular apoptosis, changes receptor functions.