Apoptosis and airway inflammation in asthma.
作者： Antonio M. Vignola , Giuseppina Chiappara , Rosalia Gagliardo , Mark Gjomarkaj , Anna Merendino
关键词: Apoptosis 、 Inflammation 、 Disease 、 Airway inflammation 、 Cell 、 Asthma 、 Immunology 、 Bronchial mucosa 、 Medicine 、 Airway Remodelling
摘要: Asthma is a disease characterized by chronic inflammation of the airways and structural alterations bron-chial tissues, often referred to as airway remodelling. The development in asthma depends upon continuous recruitment inflammatory cells from bloodstream towards bronchial mucosa their subsequent activation. It however increasingly accepted that mechanisms involved regulation survival apoptosis may play central role persistent process characterizing this disease. Increased cellular activation, enhanced cell cell:cell interactions are therefore key steps asthma, represent major causes for tissue damge, repair
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