Dysregulated splicing factor SF3B1 unveils a dual therapeutic vulnerability to target pancreatic cancer cells and cancer stem cells with an anti-splicing drug

作者: Mafficini A , Lawlor Rt , Luchini C , Jiménez-Vacas Jm , Ibáñez-Costa A

DOI: 10.21203/RS.3.RS-527836/V1

关键词: Cancer stem cellDrugSplicing factorCancer researchPancreatic cancerBiologyVulnerabilityDUAL (cognitive architecture)Text miningRNA splicing

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