作者: Marc A. Seelen , Henri G. D. Leuvenink , Rutger J. Ploeg
DOI: 10.1007/978-1-4614-4304-9_12
关键词: Inflammation 、 Lectin pathway 、 Receptor 、 Inflammatory response 、 Deceased donor 、 Intervention (counseling) 、 Immunology 、 Tissue damage 、 Innate immune system 、 Medicine
摘要: Brain death (BD) influences the early course of allografts by triggering a series nonspecific inflammatory events. A distinct role for innate immunity in initiation phase tissue damage following BD and response is increasingly appreciated. In this chapter, BD-induced inflammation will be addressed, focused on two major systems immunity—toll-like receptors (TLRs) complement.