Zinc deficiency in murine milk underlies expression of the lethal milk (lm) mutation.

摘要: The inability of nursing pups to survive on milk mice homozygous for the recessive mutation, lethal (lm), is correlated with a reduction in zinc levels both and pup carcass. Administration lmlm dams reduces observed mortality morbidity. It suggested that lm alters transport from maternal blood its study may provide useful information understanding rare human disease, acrodermatitis enteropathica.