Alternative lengthening of telomeres, telomerase, and cancer
作者： Roger R. Reddel
DOI: 10.1016/S0304-3835(02)00702-4
关键词: Biology 、 Telomere 、 Cancer research 、 Senescence 、 Werner syndrome 、 Telomerase reverse transcriptase 、 Cancer cell 、 Telomerase 、 Dyskeratosis congenita 、 Molecular biology 、 Cancer
摘要: Telomere length may be maintained in cancer cells by telomerase or an alternative lengthening of telomeres (ALT) mechanism. Low levels of telomerase activity have been …
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