Role of voltage-dependent K+ and Ca2+ channels in coronary electromechanical coupling: Effects of metabolic syndrome

作者: Zachary C. Berwick

DOI:

关键词: Katp channelsCa2 channelsCoronary vasoconstrictionMetabolic syndromeFunctional expressionInternal medicineReactive hyperemiaElectromechanical couplingExperimental biologyMedicineEndocrinology

摘要: 1. Berwick ZC, Kroessel T, Haub E. Reactions of 2,6-Dihydroxybenzoic Acid. Indiana University Undergraduate Research Conference Journal. 2007. 2. Lynch B, Payne GA, Dick G, Sturek M, Tune JD. Contribution adenosine A2A and A2B receptor subtypes to coronary reactive hyperemia: Role KV KATP channels. FASEB J. 2010. 3. Bender SB, Laughlin MH, Functional expression P2Y1 purinergic receptors in the circulation. 4. Kurian MM, Kohr MC, Moberly SP, IKCa Channels Control Coronary Blood Flow. 2011. 5. Svendsen Mather KJ, Intracoronary infusion Glucagon-like peptide 1 acutely enhances myocardial glucose uptake during ischemia canines. 6. GM, Goodwill AG, CaV1.2 channels microvascular dysfunction metabolic syndrome. In press. 7. Morrical E, voltage-dependent potassium calcium pressure-flow autoregulation. 8. Cardiac responses intravenous glucagon-like are impaired 9. Lai X, Witzmann FA, Augmented vasoconstriction epicardial perivascular adipose tissue PROFESSIONAL MEMBERSHIPS 2005 American Chemical Society 2009 Physiological 2010 Heart Association 2011 Microcirculatory for Experimental Biology Medicine ACADEMIC ASSOCIATIONS AND AWARDS 2006 President IUS Field Club 2007 Pinnacle Honors Student Government Senator Graduate Representative Cellular Integrative Physiology IU Department Travel Fellowship School Organization IUPUI Professional Theodore J.B. Stier Excellence Outstanding Abstract Award Gill Symposium Thesis honorable mention 2012 Burton Sobel Young Investigator

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