Simulating cholinesterase inhibition in birds caused by dietary insecticide exposure

摘要: Abstract We describe a stochastic simulation model that simulates avian foraging in an agricultural landscape to evaluate factors affecting dietary insecticide exposure and predict post-exposure cholinesterase (ChE) inhibition. To the model, we simulated published field studies found predictions of decay ChE inhibition reasonably approximated most observed results. Sensitivity analysis suggested location usually influenced more than diet preferences or daily intake rate. Although organophosphorus insecticides caused greater carbamate insecticides, toxicity appeared only moderately important. When impact heavy applications during breeding seasons 15 wild bird species, mean maximum species exceeded 20% at some point. At this level inhibition, birds may experience nausea and/or exhibit minor behavioral changes. Simulated risk peaked April–May August–September was lowest July. increased with proportion vegetation diet. This ones like it, help sublethal grassland animals, thereby reducing dependence ecological assessments on alone.