VII. Correlation of electrocardiographic and pathologic findings in lateral infarction

摘要: Abstract Infarction of the lateral wall left ventricle was demonstrated pathologically in 105 cases, which represents an incidence 65 per cent a series 161 cases. The cases anterolateral infarction and those posterolateral have been analyzed previous reports present study concerned with correlation electrocardiographic pathologic findings twenty-seven primary infarction. These were classified into three groups, according to distribution lesion at autopsy: (A) high, (B) low, (C) midlateral 1. A. High infarction, involving chiefly basal one-half wall, but continuing for variable distance apical one-half, found fourteen infarct limited subepicardial layer one case manifested by normal QRS complexes deeply inverted T waves typical pericarditis. Despite fact that high transmural five subendocardial other eight, it diagnostic QR pattern Lead V 5 or 6 only case. rarity abnormal Q Leads this group contrasted sharply their frequency association one-third ascribed infarcts generally spared most all wall. On hand, aV L yielded considered two strongly suggestive suspicious four Standard I not adequate substitute because failed show initial downstroke eleven patients , greater negativity right than arm. Signs customary precordial arm leads constitute indication exploration upper precordium axilla. intersection horizontal line through sternal terminus third intercostal space vertical lines plane Positions 3, 4, 5, obtained on who followed autopsy. In case, equivocal, suggestive, pathognomonic taken conjunction aided establishing diagnosis localizing position infarct. 2. B. Low largely entirely confined eight these. Abnormal and/or patterns others. electrocardiogram negative conformed Wood, Wolferth, Bellet RS-T depression 4 could be correlated acute involved mid-zones, layer, absence explained patchy character lesion. A similar recorded hours after onset pain subsequently replaced complex 3. C. Small mid-lateral infarcts, middle resembling both produced combination ventricular hypertrophy digitalis action may due small size failure take axillary leads. QRS-T abnormalities more first leads, anteroseptal actually manifestation Transmission potential variations infarcted facilitated marked counterclockwise rotation these This situation opposite previously reported where as result clockwise sufficient cause reference