作者: Kenneth Rosenfield , Robert Schainfeld , Ann Pieczek , Laura Haley , Jeffrey M Isner
DOI: 10.1016/S0735-1097(96)00498-6
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摘要: Abstract Objectives. We sought to determine the basis for restenosis within superficial femoral arteries (SFAs) and hemodialysis conduits treated with balloon-expandable stents. Background. Use of stents coronary peripheral vessels continues increase exponentially. The mechanism placed at various vascular sites is not well understood. In particular, implications deploying a stent in compressible site are Methods. After serendipitous detection deformation during intravascular ultrasound (IVUS) examination restenosed dialysis fistula, we evaluated consecutive series patients sites, including SFA (six patients) fistulae (five patients). Clinical, angiographic IVUS examinations were performed evaluate mechanisms restenosis. Results. Stent compression was identified as principal cause all SFAs. deformity reliably by angiography; however, two forms: eccentric deformation, implicating two-point compressive force, complete circumferential encroachment struts around catheter, suggesting multidirectional force. Despite redilation, secondary resulting from recurrent recurred most sites. Conclusions. Restenosis endovascular may occur result compression, phenomenon readily detected IVUS, but often angiography. These findings have significant use subject extrinsic such conduits, adductor canal segment carotid arteries. (J Am Coll Cardiol 1997;29:328–38)