Enhanced Expression of the 27 Da Heat-Shock Protein During Dena-Induced Hepatocarcinogenesis in Rat and in Human Neoplastic and Nonneoplastic Liver Tissues
作者： N. Mairesse , M. Delhaye , B. Gulbis , P. Galand
DOI: 10.1007/978-3-642-79215-1_16
关键词:
摘要: Mammalian cells respond to various kinds of stress by altering their normal patterns protein synthesis and dramatically increasing amino acid incorporation into a series specific polypeptides called “heat shock” or “stress” proteins (Hickey Weber, 1982; Landry et al., 1982a; Welch, 1993) that seem transiently protect the cell against further (Landry 1982b).
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