p53 and regulation of DNA damage recognition during nucleotide excision repair
作者： Shanthi Adimoolam , James M. Ford
DOI: 10.1016/S1568-7864(03)00087-9
关键词:
摘要: In response to a variety of types DNA damage, the p53 tumor suppressor gene product is activated and regulates number downstream cellular processes such as cell cycle arrest, apoptosis repair. Recent discoveries concerning regulation repair by p53, nucleotide excision (NER) base (BER) have paved way for studies understand mechanisms governing p53-dependent Although several theories been proposed, accumulating evidence points transcriptional regulatory role in NER, mediating expression global genomic (GGR)-specific damage recognition genes, DDB2 XPC. BER, more direct has potentially acting through protein-protein interactions with BER specific factors. These advances greatly enhanced our understanding this review comprehensively summarizes current opinions on
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