作者: F. Coraim , H. Kassal , G. Pauser , F. Stellwag , E. Wolner
DOI: 10.1007/978-3-642-69638-1_17
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摘要: Cardiogenic shock in the postoperative period of cardiac surgery still seems to be a life-threatening complication despite counterpulsation and potent pharmacological treatment [17, 22, 27, 30, 31, 33]. Effective catecholamine medication usually increases myocardial oxygen consumption [4,15,17,18,22,27,30,33]. Focal necroses within structure can sometimes detected after [24, 25, 27,28] tachycardia may also increase [4,17,18,22,29–31, Hypoxia, acidosis, ischemia as result hemodynamic deterioration will seen almost all organs [10,11,13,14]. These unfavorable conditions are most deleterious splanchnic region [9,12,19–21, 29]. They produce toxic peptides like depressant factor (MDF) lysosomal enzymes [1,2,6-9,12,19-21,26,29,32]. seem humoral component state, whose effect is considered only with regard hemodynamics. Counterpulsation lowers by reducing afterload coronary blood flow, especially diastole [2,27, 33, 34]. Nevertheless remain circulation [4, 6, 7], paralyze peripheral vascular compartment [4,6,7,27]. at this point produces no [2,4–7]. The resulting circulus vitiosus lead further [11].