Hypothalamic control of adrenaline secretion in response to insufficient glucose

摘要: 1. The rise of plasma glucose consequent upon the release adrenaline following systemic administration 3-O-methylglucose is prevented by previous infiltration with lignocaine lateral hypothalamic area on both sides. 2. similarity between this effect and that same procedure in preventing hypersecretion gastric acid response to stimulus, together known fact hypoglycaemia or some analogues produce adrenal medullary hypersecretion, provides strong presumptive evidence there exists a chemoreceptor which controls adrenaline. cells receptor like those controlling secretion, are activated an insufficiency metabolizable glucose.