作者: Simon D. Wagner
DOI: 10.1182/BLOOD-2016-04-708180
关键词:
摘要: In this issue of Blood , [Aguilar-Hernandez et al][1][1][2] refine our understanding the mechanism action interleukin 4 (IL-4) in promoting B-cell receptor (BCR)-mediated signaling primary human chronic lymphocytic leukemia (CLL) and resistance to a Bruton tyrosine kinase inhibitor (BTKi