IL-4, IgM, and resistance to BTKi and PI3Kδi in CLL.

作者: Simon D. Wagner

DOI: 10.1182/BLOOD-2016-04-708180

关键词:

摘要: In this issue of Blood , [Aguilar-Hernandez et al][1][1][2] refine our understanding the mechanism action interleukin 4 (IL-4) in promoting B-cell receptor (BCR)-mediated signaling primary human chronic lymphocytic leukemia (CLL) and resistance to a Bruton tyrosine kinase inhibitor (BTKi

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