DOI: 10.1007/978-94-007-6606-8_6
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摘要: The emission of biogenic volatile organic compounds (BVOCs), including isoprenoid compounds, methanol and oxygenated is controlled by both the existing metabolic potential a leaf gene expression responses that modulate to increase or decrease compound biosynthesis rate. This capability respond instantaneously in long term environmental variation provides plants with flexibility their adaptions biotic abiotic stresses, which are also encountered short long-term time frames. chapter reviews mechanistic basis immediate controls BVOC emissions light, temperature, ambient CO2 O2 concentrations, as well genetic involve changes patterns. Photosynthesis ultimately carbon for production, though under non-stressed conditions photosynthetic rate itself rarely so low it limits emissions. However, various pathways compete substrates produced from photosynthate, cytosolic pathways, such mevalonic acid (MVA) pathway chloroplastic 2-C-methyl-D-erythritol 4-phosphate/1-deoxy-D-xylulose 5-phosphate (MEP/DOXP). Controls over use substrate regulated among these through feedback mechanisms, specificity transport metabolites across organelle membranes, channeling NADPH reductant ATP specific steps pathways. emphasizes interactive provide major explanation longer-term physiological Emissions several types considered, isoprenoids, methanol, green volatiles aldehydes ketones.