Partial myocardial fibrosis in hypertrophic cardiomyopathy demonstrated by 18F-fluoro-deoxyglucose positron emission tomography and multislice computed tomography.
作者： Nobusada Funabashi , Keiichi Nakagawa , Issei Komuro
DOI: 10.1016/J.IJCARD.2005.01.069
关键词:
摘要: A 53-year old female with hypertrophic cardiomyopathy (HCM) was undergoing transthoracic echocardiography annually. recent echocardiogram (TTE) revealed reduced motion of the apical inter-ventricular septum (IVS) left ventricle (LV). To evaluate viability myocardium, positron emission tomography (PET) (Advance NXi, General Electric, Milwaukee, WI) performed after oral glucose loading using F-fluorodeoxyglucose and myocardial uptake in anterior IVS, suggesting partially necrotic tissue (arrowheads Fig. 1). coronary artery electrocardiogram (ECG)-gated enhanced multislice computed (CT) (Light Speed Ultra 16, intravenous injection iodinated contrast, a 0.625-mm slice thickness, helical pitch 3.25. CT scanning retrospective ECG-gated reconstruction at 30 s (early phase) 8 min (late phase). Axial source images normal arterial lumen without calcification, absence heart disease that could cause partial necrosis IVS LV. Also, hypertrophy The sections lower intensity than other LV early phase (arrowheads, 2A,B), corresponding site 1 represented by PET.
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