摘要: Much research about memory in autism concerns the hypothesis that is similar to adult-onset amnesia. Initial support for came from post-mortem studies of individuals with showing abnormalities hippocampus and related brain structures, as well behavioral finding contrasts between intact cued recall impaired free recognition autism. The was later brought into question by performance on explicit tasks typically present paper proposes a possible reconciliation these contradictory findings, suggesting there selective damage limbic-prefrontal episodic system, sparing limbic-only perceptual representation semantic system. This view consistent other evidence early systems involving cooperation limbic system medial prefrontal cortex