作者: Antonis S. Manolis , Antonis A. Manolis
DOI: 10.1007/978-3-319-73764-5_6
关键词:
摘要: The right ventricle (RV) responds differently to prolonged exhaustive and competitive exercise, usually with enlargement, than does the left ventricle. Evidence has accumulated indicating that regular intense endurance exercise sporting can promote electrical structural remodeling of RV, leading fibrosis. This “exercise-induced cardiomyopathy” mimics features observed in arrhythmogenic ventricular dysplasia/cardiomyopathy (ARVD/C). In addition, exercise-induced impairment atrial function (atrial remodeling), mostly atrium related RV systolic dysfunction, also been suggested which favors development arrhythmias. It should be noted these adverse effects seem limited extreme intensity levels not recreational or moderate levels. These deleterious heavy on heart studied by several methods, including standard newer echocardiographic techniques, stress testing ECG, cardiac magnetic resonance imaging, biomarkers, electroanatomical mapping and/or an electrophysiology study, specific cases genetic testing. All issues are reviewed this chapter.