作者: Maram Zakko , Roberto Groszmann
DOI: 10.1007/978-1-4612-1808-1_29
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摘要: In portal hypertension, the increase of pressure is due to both an in vascular resistance and splanchnic blood flow (1–3). Prehepatic, intrahepatic, posthepatic sites increased venous may result hypertension (1). Prehepatic are caused by obstruction hepatic veins, respectively. Intrahepatic be anatomic alterations lobule with secondary compression venules, sinusoids, functional derangement intrahepatic microcirculation.