In utero programming of cardiovascular disease
作者： D.J.P. Barker
DOI: 10.1016/S0093-691X(99)00258-7
关键词:
摘要: Low birth weight, thinness and short body length at are now known to be associated with increased rates of cardiovascular disease non-insulin dependent diabetes in adult life. The fetal origins hypothesis proposes that these diseases originate through adaptations which the fetus makes when it is undernourished. These may cardiovascular, metabolic or endocrine. They permanently change structure function body. Prevention depend on prevention imbalances growth between pre- post-natal growth, nutrient supply fetus.
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