摘要: Progenitor cells have the capability to home myocardium in response ischemia. Cell adhesion markers, particular integrins, play an important role trafficking of stem myocardium. In addition, damaged secretes several chemokines and growth factors that recruit these precursor heart. Nitric oxide synthase hormones can also contribute progenitor The recruitment ischemic is a complex interchange between cell chemokines, better understanding processes may lead more efficient use for therapeutic benefit.