作者: David G. Benditt , William Fabian , Demos Iskos , Keith G. Lurie
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摘要: Vasovagal syncope is characterized by transient failure of usually reliable physiologic mechanisms responsible for maintaining both systemic arterial pressure and cerebral blood flow. Two circulatory phenomena are almost universally present: vasodilation bradycardia. A third phenomenon, cerebrovascular constriction, has also been described but its contribution to the faint less well established. The neural reflex pathways triggering changes in vasovagal incompletely understood, have recently subject renewed interest. In part, this interest probably stems from frequency with which symptoms now recognized be cause fainting spells. Additionally, however, there an increasingly need develop treatment strategies those affected patients whom recurrent particularly troublesome. It goal discussion focus on aspects control, particular potential interactions among certain humoral systems, may contribute inappropriate responses associated faint.