作者: Ioannis E. Koutroubakis , Aekaterini Sfiridaki , Georgia Tsiolakidou , Constantina Coucoutsi , Angeliki Theodoropoulou
DOI: 10.1097/MEG.0B013E3282FAA759
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摘要: BackgroundPatients with inflammatory bowel disease (IBD) have an increased risk of thromboembolic events. Imbalance fibrinolysis has been suggested as one the possible pathogenetic mechanisms. As plasminogen activator inhibitor-1 (PAI-1) and thrombin-activatable inhibitor (TAFI) a