作者: Guzal Khayrullina , Sara Bermudez , Kimberly R. Byrnes
DOI: 10.1186/S12974-015-0391-8
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摘要: Background Spinal cord injury (SCI) results in the activation of NADPH oxidase (NOX) enzyme, inducing production reactive oxygen species (ROS). We hypothesized that NOX2 isoform plays an integral role post-SCI inflammation and functional deficits.