作者: Masanori Asakura , Yasunori Ueda , Osamu Yamaguchi , Takayoshi Adachi , Atsushi Hirayama
DOI: 10.1016/S0735-1097(01)01135-4
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摘要: Abstract OBJECTIVES To test our hypothesis that the development of vulnerable plaques is not limited to culprit lesions, but a pan-coronary process, we directly observed all three major coronary arteries by angioscopy and evaluated prevalence yellow in patients with myocardial infarction (MI). BACKGROUND Although pathologic studies have suggested disruption atheromatous plaque plays role acute MI, whole MI has been clarified. METHODS Thirty-two undergoing follow-up catheterization one month after onset were prospectively consecutively enrolled this study. The thrombus was successfully 20 (58 arteries, 21 lesions) angioscopy. diameter stenosis (DS) lesions maximal (maxDS) nonculprit segments angiographically measured for each artery. RESULTS DS maxDS 27 ± 17% 19 13%, respectively. Yellow detected (90%) 17 (81%) equally prevalent infarct-related non–infarct-related (3.7 1.6 vs. 3.4 1.8 plaques/artery). However, only (2%) CONCLUSIONS In are widely diseased multiple though nondisrupted plaques. Acute may represent process development.