The immune system and murine atherosclerosis.

摘要: In this review the modulation of lipoprotein metabolism and atherogenesis by innate adaptive immune systems mouse is discussed. While recognizing participation all components system in atherogenesis, it clear that robust may proceed without an response. But even when are active, outcome reflects a balance between pro- anti-inflammatory reactants cells. This network interactions summarized complemented other reviews series. Also noted differential response different vascular beds following manipulation system. Further work required to achieve fuller understanding role these especially with prospect favorably modulating participating as for example vaccination approach.