作者: Wim H. J. Vanhouten , Sally A. Mackenzie
DOI: 10.1007/978-1-4615-4235-3_8
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摘要: Cytoplasmic male sterility (CMS) is the condition in which a plant unable to shed viable pollen as consequence of mitochondrial mutations. The dominant mutations described cause CMS various crop species can be compensated for by action nuclear fertility restorer (suppressor) genes, thus providing an important system study nuclear-mitochondrial interactions (Hanson, 1991; Schnable and Wise, 1998). In all cases studied so far, CMS-associated create novel open reading frames (ORFs) (Dewey et al., 1986; Young Hanson, 1987; Kohler Laver Singh Brown, Johns 1992; Krishnasamy Makaroff, 1993). translation products from these ORFs presumably interfere with development, although mechanisms this disruption occurs are still unknown.