Factors Associated With Slow Progression in Huntington's Disease

作者: R. H. Myers , D. S. Sax , W. J. Koroshetz , C. Mastromauro , L. A. Cupples

DOI: 10.1001/ARCHNEUR.1991.00530200036015

关键词:

摘要: … In addition, factors related to disease progressioncan be controlled for in clinical trials. The assessment ofrate of disease pro¬ gression was limited to persons fol¬ lowed up for at least 3 …

参考文章(15)
N C Terrin, R H Myers, R B D'Agostino, P A Wolf, N Goldmakher, M Schoenfeld, L A Cupples, Maternal factors in onset of Huntington disease. American Journal of Human Genetics. ,vol. 37, pp. 511- 523 ,(1985)
P M Conneally, L A Farrer, A genetic model for age at onset in Huntington disease. American Journal of Human Genetics. ,vol. 37, pp. 350- 357 ,(1985)
M. Flint Beal, Neil W. Kowall, David W. Ellison, Michael F. Mazurek, Kenton J. Swartz, Joseph B. Martin, Replication of the neurochemical characteristics of Huntington's disease by quinolinic acid Nature. ,vol. 321, pp. 168- 171 ,(1986) , 10.1038/321168A0
R. H. Myers, J. P. Vonsattel, T. J. Stevens, L. A. Cupples, E. P. Richardson, J. B. Martin, E. D. Bird, Clinical and neuropathologic assessment of severity in Huntington's disease Neurology. ,vol. 38, pp. 341- 341 ,(1988) , 10.1212/WNL.38.3.341
R Schwarcz, W. Whetsell, R. Mangano, Quinolinic acid: an endogenous metabolite that produces axon-sparing lesions in rat brain Science. ,vol. 219, pp. 316- 318 ,(1983) , 10.1126/SCIENCE.6849138
D. Lovinger, G White, F. Weight, Ethanol inhibits NMDA-activated ion current in hippocampal neurons Science. ,vol. 243, pp. 1721- 1724 ,(1989) , 10.1126/SCIENCE.2467382
O.J.S. Buruma, W. Van der Kamp, E.C. Barendswaard, R.A.C. Roos, D. Kromhout, E.A. Van der Velde, Which factors influence age at onset and rate of progression in Huntington's disease? Journal of the Neurological Sciences. ,vol. 80, pp. 299- 306 ,(1987) , 10.1016/0022-510X(87)90164-X