Uterine activity: implications for the condition of the fetus.

作者: Petra C.A.M. Bakker , Herman P. van Geijn

DOI: 10.1515/JPM.2008.003

关键词:

摘要: Aim: The aim of this review is to provide more insight in the fetal mechanisms as a response uterine contractions and emphasize importance correct assessment activity (UA) patterns during labor. Study results: UA causes decreased flow through artery. In healthy uncompromised fetus, will not cause acidemia. fetus has developed certain protection survive labor; (1) During contraction, preload increases enables maintain constant blood umbilical artery (2) middle cerebral artery, i.e., brain sparing effect. shortcoming those compromised case excessive risk adverse outcome. effect become pronounced compensate for oxygen saturation. Maintenance normal UA, especially sufficiently long relaxation time, essential so that supply well oxygenated maternal intervillous space be restored saturation can remain stable. Conclusion: Adequate monitoring prerequisite proper reading interpretation cardiotocograms. It alarms cases help prevent Uterine contraction deserves full attention daily obstetric practice. © 2008 by Walter de Gruyter.

参考文章(38)
H.-B. Krebs, R.E. Petres, L.J. Dunn, Intrapartum fetal heart rate monitoring. VIII. Atypical variable decelerations. American Journal of Obstetrics and Gynecology. ,vol. 145, pp. 297- 305 ,(1983) , 10.1016/0002-9378(83)90714-7
David G. Chaffin, Caroline F. Anderson, Kathryn L. Reed, Dana P. Damron, Changes in umbilical arterial and venous blood flow velocity waveforms during late decelerations of the fetal heart rate. Obstetrics & Gynecology. ,vol. 84, pp. 1038- 1040 ,(1994)
F Oberheuser, R Grosspietzsch, L V Klitzing, F Klink, Uterine contraction intervals and transcutaneous levels of fetal oxygen pressure Obstetrics & Gynecology. ,vol. 57, pp. 437- 440 ,(1981)
S. YAGEL, E. ANTEBY, Y. LAVY, A. BEN CHETRIT, Z. PALTI, D. HOCHNER-CELNIKIER, M. RON, Fetal middle cerebral artery blood flow during normal active labour and in labour with variable decelerations. British Journal of Obstetrics and Gynaecology. ,vol. 99, pp. 483- 485 ,(1992) , 10.1111/J.1471-0528.1992.TB13786.X
Tekoa King, Julian Parer, The physiology of fetal heart rate patterns and perinatal asphyxia. Journal of Perinatal & Neonatal Nursing. ,vol. 14, pp. 19- 39 ,(2000) , 10.1097/00005237-200012000-00003
Hui Li, Saemundur Gudmundsson, Per Olofsson, Acute centralization of blood flow in compromised human fetuses evoked by uterine contractions. Early Human Development. ,vol. 82, pp. 747- 752 ,(2006) , 10.1016/J.EARLHUMDEV.2006.03.011
Hui Li, Saemundur Gudmundsson, Per Olofsson, Acute increase of umbilical artery vascular flow resistance in compromised fetuses provoked by uterine contractions. Early Human Development. ,vol. 74, pp. 47- 56 ,(2003) , 10.1016/S0378-3782(03)00084-7
Christine East, Kimble Dunster, Paul Colditz, Fetal oxygen saturation and uterine contractions during labor. American Journal of Perinatology. ,vol. 15, pp. 345- 349 ,(1998) , 10.1055/S-2007-993955
Dick K. Donker, Herman P. van Geijn, Arie Hasman, Interobserver variation in the assessment of fetal heart rate recordings. European Journal of Obstetrics & Gynecology and Reproductive Biology. ,vol. 52, pp. 21- 28 ,(1993) , 10.1016/0028-2243(93)90220-7
P.C.A.M. Bakker, P.H.J. Kurver, D.J. Kuik, H.P. Van Geijn, Elevated uterine activity increases the risk of fetal acidosis at birth. American Journal of Obstetrics and Gynecology. ,vol. 196, pp. 313- ,(2007) , 10.1016/J.AJOG.2006.11.035