作者: Alan D. Dodge
DOI: 10.1016/B978-0-08-029224-3.50012-5
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摘要: Abstract The damaging effect of toxic oxygen species in plants is minimised by a number protective mechanisms. These include superoxide dismutases for superoxide, catalase and ascorbate peroxidase hydrogen peroxide, carotenoids singlet triplet chlorophyll, α-tocopherol free radicals such as hydroxyl. Many herbicides exert their action overtaxing or destroying these systems. bipyridylium are reduced chloroplast electron transport to univalent radical which reoxidized molecular generate the anion radical. Hydroxyl produced from peroxide Haber-Weiss type reaction, paraquat diphenyl ether herbicide oxyflurofen may also function similar manner. photosynthetic inhibitor herbicides, operate at one major site action. When flow inhibited, unquenched chlorophyll passes longer lived state. Interaction between generates oxygen. A both hydroxyl on unsaturated fatty acids membrane lipids induction peroxidation reactions. lead destruction thus cellular dis-organisation death. potential generation enhanced prevent formation carotenoid pigments inhibiting desaturase reactions phytoene phytofluene, cyclisation lycopene.