摘要: Mycobacterium leprae must adopt a metabolic strategy and undergo various alterations upon infection to survive inside the human body for years in dormant state. A change lipid homeostasis is highly pronounced leprae. Lipids play an essential role survival pathogenesis of mycobacteria. are present several forms serve multiple roles from being source nutrition, providing rigidity, evading host immune response serving as virulence factors, etc. The synthesis degradation lipids regulated process key future drug designing diagnosis In current review, account distinct served by lipids, mechanism their has been elucidated.