Genetic analysis and molecular resistance to race 2 of Fusarium wilt in pigeonpea [Cajanus cajan (L.) Millsp.]

摘要: Abstract The long-stagnated productivity of pigeonpea may be attributed to its vulnerability several diseases which vascular wilt caused by Fusarium udum Butler is paramount significance. Disease management approaches that rely on chemicals are not very appropriate especially in view rising environmental concerns and resource poor nature marginal farmers. Therefore, deploying host plant resistance genetic improvement schemes through molecular tagging underlying genes central breeding. To this end, here we offer SSR markers for race 2 (FW) KPL 43 (resistant) bulked segregant analysis (BSA). disease incidence data 271 F individuals derived from Bahar (susceptible) × KPL revealed the involvement two or more recessive (1:15) with duplicate dominant epistasis. Marker polymorphism survey using 270 hypervariable pigeonpea-specific (HASSRs) yielded 115 polymorphic (42.6%) between parents. HASSR8 190 , HASSR58 180 HASSR121 170 differentiated resistant susceptible bulks. A joint segregation each marker FW population supported linkage SSRs. Recessive class (RCA) based genotypic ten individual also confirmed SSRs gene. independent assortments was observed pair associated markers, corroborated alignment these three different chromosomes genome. association further validated a panel 89 genotypes single (SMA) Kruskal Wallis test. Given conferring resistance, have tremendous implications breeding genotypes.