作者: D. R. Campion , G. J. Hausman
DOI: 10.1007/978-94-009-1395-0_20
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摘要: We have highlighted the two models (fetal hypophysectomy and obesity) to illustrate that: 1) histochemical biochemical differences associated with do not express themselves until 72 74d of gestation that myonuclear proliferation (DNA content satellite cell content) are influenced. Thus, muscle DNA fiber numbers under neural or hormonal (endocrine) regulation after 45d gestation; secondly, 2) in obese models, genetic difference from lean controls number must be regulated by events occurring before 70 90 d 3) paracrine autocrine fetal growth is indicated similarity serum proliferative activity across ages models; 4) since preadipocytes respond differently sera these terms activity, cells different developmental controls. adipose tissue relatively more responsive extratissue stimuli while resistent such stimuli. To extrapolate further, data imply one could influence fat independent an effect on myogenesis.