Chapter 83 – Exercise-induced Cardiac Arrhythmias

作者: Ruey J. Sung , Chi Tai Kuo , Wen Ter Lai

DOI: 10.1016/B0-7216-0323-8/50086-5

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摘要: Exercise-induced cardiac arrhythmias are that occur during or after physical exertion. Clinical consequences of these range from palpitation, dizziness, syncope, angina, acute myocardial infraction, and congestive heart failure to even sudden death. 1 2 may be observed in patients prone ischemia such as those with atherosclerotic disease primary secondary cardiomyopathies. However, they also seemingly healthy individuals all ages. In the latter population, exercise-induced can benign, but malignant due electrical structural abnormalities acquired (e.g., drug-induced) congenital long QT syndrome arrhythmogenic right ventricular dysplasia). The heterogeneity underlying pathophysiologic conditions has made diagnosis management a clinical challenge.

参考文章(35)
Ruey J. Sung, Michael R. Lauer, Polymorphic Ventricular Tachycardia and Long QT Syndromes Fundamental Approaches to the Management of Cardiac Arrhythmias. pp. 731- 761 ,(2000) , 10.1007/978-94-011-4371-4_16
Qiuyun Chen, Glenn E. Kirsch, Danmei Zhang, Ramon Brugada, Josep Brugada, Pedro Brugada, Domenico Potenza, Angel Moya, Martin Borggrefe, Günter Breithardt, Rocio Ortiz-Lopez, Zhiqing Wang, Charles Antzelevitch, Richard E. O'Brien, Eric Schulze-Bahr, Mark T. Keating, Jeffrey A. Towbin, Qing Wang, Genetic basis and molecular mechanism for idiopathic ventricular fibrillation Nature. ,vol. 392, pp. 293- 296 ,(1998) , 10.1038/32675
Douglas P. Zipes, José Jalife, Cardiac Electrophysiology : From Cell to Bedside ,(1990)
Gisèle Bonne, Lucie Carrier, Pascale Richard, Bernard Hainque, Ketty Schwartz, Familial Hypertrophic Cardiomyopathy: From Mutations to Functional Defects Circulation Research. ,vol. 83, pp. 580- 593 ,(1998) , 10.1161/01.RES.83.6.580
Heikki Swan, Kirsi Piippo, Matti Viitasalo, Päivi Heikkilä, Timo Paavonen, Katariina Kainulainen, Juha Kere, Pekka Keto, Kimmo Kontula, Lauri Toivonen, Arrhythmic disorder mapped to chromosome 1q42–q43 causes malignant polymorphic ventricular tachycardia in structurally normal hearts Journal of the American College of Cardiology. ,vol. 34, pp. 2035- 2042 ,(1999) , 10.1016/S0735-1097(99)00461-1
Antoine Leenhardt, Vincent Lucet, Isabelle Denjoy, Francis Grau, Dien Do Ngoc, Philippe Coumel, Catecholaminergic Polymorphic Ventricular Tachycardia in Children A 7-Year Follow-up of 21 Patients Circulation. ,vol. 91, pp. 1512- 1519 ,(1995) , 10.1161/01.CIR.91.5.1512
A. Rampazzo, A. Nava, P. Erne, M. Eberhard, E. Vian, P. Slomp, N. Tiso, G. Thiene, G. A. Danieli, A new locus for arrhythmogenic right ventricular cardiomyopathy (ARVD2) maps to chromosome 1q42–q43 Human Molecular Genetics. ,vol. 4, pp. 2151- 2154 ,(1995) , 10.1093/HMG/4.11.2151
Jeffrey A Towbin, Matteo Vatta, Molecular biology and the prolonged QT syndromes The American Journal of Medicine. ,vol. 110, pp. 385- 398 ,(2001) , 10.1016/S0002-9343(00)00715-4