摘要: The lesions described in this chapter result from anomalous cortical development dating to the fetal period. Only those sufficiently severe impede survival are commonly observed infants or children; many others survived with various degrees of functional impairment and found at autopsy adults. During nineteenth century dysplasias cerebral cortex were generally not distinguished residual due loss parenchyma scarring. first step direction was taken by Bresler (1899) who differentiated scarring ulegyria maldevelopment polymicrogyria, which he called “microgyria”. term microgyria had been used earlier for small gyri any type; practice carried on least a decade after Bresler’s report and, sporadically, into more recent literature. There is some overlap usage names given specific types dysplasias, but reflects mainly individual semantics rather than disagreement about basic features identity lesions.