Developmental mechanisms of heart disease

作者: Robert M. Hamilton

DOI: 10.1097/00003246-199701000-00040

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摘要: Introduction: Developmental Cardiology Comes of Age Section I Molecular Biology: Overview: Analysis Cardiac Phenotypes: Problems, Progress and Prospects Retinoblastoma Pocket Proteins the Terminally Differentiated State Muscle Cells Regulation Cardiomyogenic Differentiation Divsification Positive Negative Promoter Elements Regulating @-Smooth Actin Gene Expression in Embryonic Myocytes The Role Serum Response Factor, Ying Yang One, Basic-Helix Loop-Helix Factors on @-Actin During Early Skeletal Development Troponin C Genes Analyzed at mRNA Protein Levels Developing Muscles Cardiac-Specific Homebox-Containing Mouse II Clinical Application Biology to Medical Genetics Genetic Hypertrophic Cardiomyopathy Japan Biochemical Characterization X-Linked Dilated Morphometric Study Right Ventricular Hypetrophy with Congenitally Malformed Hearts Myosin Heavy Chain Various Blood Vessels Organs Changes Rabbit Ductus Arteriosus Smooth Exposure Cyclic Adenosine Monophosphate III Cardiogenesis: Formation Morphogenesis Primary Heart Tube Proper Laboratory Practice Experimental Studies Abnormal Cardiovascular Extracellular Matrix, Receptors, Basis for Segmental Pattern Cushion Mesenchyme Formation: ES/130 Emryonic Chick Effect Endoderm Growth Precardiac Mesoderm Vitro Interactions Between Mesenchymal Matrix Rat Immunohistochemical Localization Platelet-Derived Factor (PDGF) Embryos Fetuses HeartDevelopment by Fibroblast Factor-2 System A Preview Neural Crest Contribution Conotruncus Its Septation Advert Era Normal Outflow Tract Atrail Septal Complex Human Heart: Spina Vestibuli Topography Embryo: From Function Organization Specilized Myocardium Maternal Thp Fetuses: Enlarged Malformations Vasculogenesis Embryo Coronary Artery Distribution Cells, Fibronectin, Tenascin Incubation Day 5 Late Fetal Retinoic Acid-Induced Transposition Great Arteries: Morphology, Physiology, Biochemistry Asymmetry Inferior Superior Atrioventricular Endocardial Cushions Mechanisms Complete Arteries Produced All-Trans Acid Conduction as Determined Leu-7 Immunohistochemistry Scanning Electron Microscopy Three-Dimensional Reconstruction HNK-1 Immunoreativity Codistribution Tissue Actylcholinesterase Prenatal Comparison Teratogen-Treated reactivity Lectin Staining Origin Orifice Pulmonary Vein Anomalies Induced Small Dose Bis-Diamine An Animal Model CATCH 22 Bis-Diamine-Induced Defect Malformation Functional Trichloroethylene IV Maturation Coupling Biomechanics Modeling Control Hemodynamics Characteristics Activity Sarcolemmal Na+-Ca2+ Exchanger Perinatal Contraction Arterial a High-Speed Video Camera: Presence Primitive Anterior Systemic Channel Mechanical Properties Observation F-Actin N-Cadherin Fibronectin Looping th eChick Using CLSM Bundle Loop V Models Defects: Malformation: Breaking Symmetry: Left-Right Xenopus Laevis inv Insertional Mutation Deciphering Lateralization Process or Way Understand Congenital Defects Atrioventircular Pathogenesis Common Canal: Morphological iv/iv Morphology Atrial Chambers Conotruncal Anomaly Face Syndrome Significance 22qll. 2 Deletion Detection 22qll Patients Malformations, DiGeorge, Velocardiofacial, Syndromes 22: Can Explain Phenotype? Stochastic Disease Down WKY/NCrj Strain Rate: New Abnormalities Branchial 45X Canal Mice Heterotaxia Situs Inversus Mutant Visceroatrial Heterotaxy Rodents VI Epidemiology: Epidemiologic Approach Cardiovascualr Etiologic Distinctions Case-Control Risk EUROCAT Subproject onEpidemiology Disease: First Completed Index

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