作者: Nobuharu Kosugi , Arinobu Tojo , Hitoshi Shinzaki , Tokiko Nagamura-Inoue , Shigetaka Asano
DOI: 10.1182/BLOOD.V95.6.2188.2188
关键词:
摘要: Chronic myeloid leukemia (CML) is a myeloproliferative disorder that results from the clonal expansion of pluripotent stem cell, characterized by Philadelphia chromosome (Ph).[1][1] The clinical course CML generally biphasic, representing an initial chronic phase (CP) and subsequent