作者: Jacob J. Ruzevick , Matthew Bender , Lindsay Edwards , Rachel Grossman , Ming Zhao
DOI: 10.1227/NEU.0000000000000080
关键词:
摘要: Background Vasospasm contributes to delayed cerebral ischemia after aneurysmal subarachnoid hemorrhage (aSAH). Glutamate concentrations increase aSAH and correlate with vasospasm in experimental SAH. The Hp2-2 genotype is associated higher risk of We tested the efficacy S-4-CPG, a metabotropic glutamate receptor inhibitor, for treatment SAH Hp1-1 mice.