摘要: Prostaglandin E,(PGE,) increases the deformability of red blood cells by decreasing cell volume, while PGE2has opposite effect. This takes place within seconds and requires low, physiological doses prostaglandin. They may be important in controlling capillary circulation. These effects play a role evolution shock essential hypertension. PGE has induced sickling susceptible cells. PGE,inhibits PGE2potentiates platelet aggregation variety agents. seem to modulated cyclic adenosine monophosphate levels, which are increased PGE,and decreased PGE2. Antiaggregation properties PGE,markedly increase poststorage yield functional platelets for transfusion. Erythropoiesis is stimulated both direct effect on erythroid precursor stimulating erythropoietin release from kidneys animals infused with PGE1.
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