Cardiac Toxicity of Singlet Oxygen: Implication in Reperfusion Injury
作者： M.-C. Toufektsian , F.R. Boucher , S. Tanguy , S. Morel , J.G. de Leiris
DOI: 10.1089/152308601750100506
关键词:
摘要: Oxygen-derived free radicals (O2•-, H2O2, and •OH) that are produced during postischemic reperfusion currently suspected to be involved in the pathogenesis of tissue injury. Another reactive oxygen species, electronically excited molecular (1O2), is increasing interest area experimental research cardiology. In this review discussed main potential sources singlet organism, particularly myocardium, various cardiovascular cytotoxic effects induced by intermediate, growing evidence its involvement ischemia/reperfusion
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